Pancragen (Lys-Glu-Asp-Trp): The Only Bioregulator With Primate Data

Pancragen has six published papers naming it — the largest compound-specific literature in the Khavinson family — including two studies in old rhesus monkeys. What primate data does and does not buy you.

pancragen pancragene bioregulator khavinson Lys-Glu-Asp-Trp KEDW glucose tolerance pancreas primate

Key Research Findings

  • Pancragen (KEDW, Lys-Glu-Asp-Trp) has six papers naming it — the largest compound-specific literature in the Khavinson family.
  • It is the only member with non-human-primate data: two studies in old female rhesus monkeys.
  • Rodent work centres on experimental diabetes; cell work on pancreatic cell differentiation during ageing.
  • The strength is convergence across cell, rodent and primate levels rather than depth at any one level.
  • KEDW is KED (Vesugen/Vesilute) plus one tryptophan — one residue separates best- and least-documented.
  • Formula C26H36N6O9, 576.61 g/mol — corrected from an earlier catalog value inconsistent with the sequence. No amount is established for any species.

Why Pancragen Stands Out

Pancragen — also written pancragene — is a synthetic tetrapeptide, Lys-Glu-Asp-Trp (KEDW), classified within the bioregulator family as pancreas-associated.

It has six published papers naming it directly. That is the largest compound-specific literature of any member of this family, and two of those papers are in old rhesus monkeys — the only non-human-primate work in the entire set.

Structurally it is also the most interesting comparison case available: KEDW is KED extended by a single tryptophan. One residue separates the family's best-documented compound from one with no literature at all.

Research use only. Supplied for laboratory research. Not for human or veterinary use. No marketing authorisation exists and no therapeutic claim is made.

Primate Studies

Two papers from the same group report on old female rhesus monkeys: correction of impaired glucose tolerance using the tetrapeptide (Goncharova et al., Adv Gerontol 2015, PMID 28509500) and the impact of the tetrapeptide on endocrine function of the pancreas in old monkeys (Goncharova et al., 2014, PMID 25946840).

Primate data is a genuine step up from rodent work in physiological proximity. It is not a substitute for a controlled human trial, and these are small-cohort ageing studies from one laboratory, not a programme.

Rodent and Cell Work

The rodent literature centres on experimental diabetes: an effect on blood glucose, capillary permeability and adhesion in rats with experimental diabetes mellitus (Khavinson et al., Bull Exp Biol Med 2007, PMID 18642713) and an effect on functional morphology of the pancreas in the same model (Kvetnoi et al., 2007, PMID 18225766).

At cell level, a study reports effects on the differentiation of pancreatic cells during ageing (Khavinson et al., Bull Exp Biol Med 2013, PMID 23486591). A further paper discusses prospects for correction of metabolic disorders in elderly people (Korkushko et al., 2011, PMID 22448364) — read the design carefully, since "prospects" papers in this literature are frequently discussion rather than controlled trial.

Reading the Evidence

Convergence across levels is the real strength. Cell differentiation, rodent physiology and primate glucose handling point the same direction. That is a better evidentiary shape than six papers at one level would be.

The concentration caveat still applies. The primate work is from a single group, and most of the set is from the originating programme.

Ageing models are not disease models. These are studies in old animals, not in a human metabolic condition.

No amount is established for any species or context, and none is offered here.

Structure and Handling

  • Sequence. Lys-Glu-Asp-Trp (KEDW), tetrapeptide.
  • Formula. C26H36N6O9, 576.61 g/mol.
  • Form. Lyophilised powder, ≥98% purity, third-party certificate of analysis per batch.
  • Storage. Sealed vial at −20 °C, protected from light; reconstituted solution at 2–8 °C.
  • Reconstitution. Bacteriostatic water down the vial wall, never onto the powder cake. Do not shake. See the reconstitution calculator.

The catalog record for this compound previously carried a molecular formula inconsistent with the stated sequence. It has been corrected against the sequence and verified. If you sourced material against the earlier figure, check the certificate of analysis for the batch you hold.

Vesugen/Vesilute (KED) is Pancragen minus the tryptophan and has no compound-specific literature — a useful contrast if sequence-length effects are the question. See the bioregulator overview for the full family.

Frequently Asked Questions

What is Pancragen?

Pancragen (also pancragene) is a synthetic tetrapeptide, Lys-Glu-Asp-Trp (KEDW), classified within the Khavinson bioregulator family as pancreas-associated. It has six published papers naming it directly — the largest compound-specific literature in the family.

Is there primate research on Pancragen?

Yes, and it is the only compound in this family with non-human-primate data. Two papers report on old female rhesus monkeys: correction of impaired glucose tolerance (PMID 28509500) and impact on pancreatic endocrine function (PMID 25946840). Both are small-cohort ageing studies from one laboratory.

What does the rodent research show?

Studies in rats with experimental diabetes mellitus report effects on blood glucose, capillary permeability and adhesion (PMID 18642713), and on functional morphology of the pancreas (PMID 18225766). A cell-level study addresses differentiation of pancreatic cells during ageing (PMID 23486591).

Is there an established Pancragen dosage?

No. Despite having the largest literature in the family, no controlled human programme exists and no published figure is available for any population or context.

How does Pancragen relate to Vesugen?

Pancragen is KEDW — the KED tripeptide of Vesugen and Vesilute extended by a single tryptophan residue. One residue separates the best-documented compound in this family from one with no compound-specific literature at all, which makes the pair a useful contrast if sequence-length effects are the research question.

What is the molecular formula of Pancragen?

C26H36N6O9, molecular weight 576.61 g/mol, derived from the Lys-Glu-Asp-Trp sequence and cross-checked against the residue composition. Our catalog previously carried a formula inconsistent with that sequence; it has been corrected. If you sourced material against the earlier figure, check the certificate of analysis for the batch you hold.

References

  1. Goncharova ND, Vengerin AA, Khavinson VKh. Correction of impaired glucose tolerance using tetrapeptide (Pancragen) in old female rhesus monkeys Advances in Gerontology (2015)
  2. Goncharova ND, Vengerin AA, Khavinson VKh. Impact of tetrapeptide pancragen on endocrine function of the pancreas in old monkeys Advances in Gerontology (2014)
  3. Khavinson VKh, Popovich IG, Linkova NS. Effects of pancragen on the differentiation of pancreatic cells during their ageing Bulletin of Experimental Biology and Medicine (2013)
  4. Khavinson VKh, Malinin VV, Grigoriev EI, Ryzhak GA. Effect of pancragen on blood glucose level, capillary permeability and adhesion in rats with experimental diabetes mellitus Bulletin of Experimental Biology and Medicine (2007)
  5. Kvetnoi IM, Yuzhakov VV, Konovalov SS, Khavinson VKh. Effect of tetrapeptide pancragene on functional morphology of the pancreas in rats with experimental diabetes mellitus Bulletin of Experimental Biology and Medicine (2007)
  6. Korkushko OV, Khavinson VKh, Shatilo VB, Antonyk-Sheglova IA. Prospects of using pancragen for correction of metabolic disorders in elderly people Bulletin of Experimental Biology and Medicine (2011)
Research Use Only: This content is intended for laboratory and scientific research purposes only. It is not intended for human use, medical advice, diagnosis, or treatment. All compounds discussed are for in vitro and preclinical research contexts.